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Chronic pain involves persistent nociceptor and microglial overactivation, which leads to electron transport chain (ETC) dysfunction in neuronal mitochondria and excessive production of reactive oxygen species (ROS) and reactive nitrogen species (RNS)

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In analyses of ChIP-seq data, we observed that many of these genes were devoid of NRF2 in their promoter regions

Metformin and dipeptidyl Peptidase-4 inhibitor differentially modulate the intestinal microbiota and plasma metabolome of metabolically dysfunctional mice

Values are presented as means SD (Table.1)